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In this work, we investigate the molecular mechanisms by which GLP-1, oxyntomodulin, exendin-4, and exendin-P5 bind and activate the GLP-1R using a combination of structural biology, molecular dynamics simulations, and pharmacological studies combined with extensive receptor mutagenesis

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Susan Ahern et al.

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IGF-1 Elevation as a Pharmacologic Signal CJC-1295's GHRH agonism produces dose-proportional GH elevation, which raises IGF-1